The Error of the Millennium in Veterinary Medicin
The background
Diseases of the musculoskeletal system in dogs have been a considerable
veterinary problem for decades. Statistics compiled by the canine science
associations indicate that 70 - 75% of the entire canine population are
affected. More than half of the dogs of all breeds exhibit pathological
changes, especially of the acetabulum and femur, known as hip dysplasia (HD)
or canine hip dysplasia (CHD)
The etiological or causal background to canine hip dysplasia has hitherto
generally been assumed to be multifactorial heredity. This term originates
from farm and slaughter animal breeding and postulates that not only
heredity but also environ-mental factors – especially nutrition – play a
role in determining characteristics. The relative importance of these
factors is expressed as the degree of heritability. For CHD, percentage
values of up to 60 % have been assumed or, with reference to 1, of 0.2 to
0.6.
The dog breeding associations in the United States, United Kingdom, France
and Germany developed X-ray systems designed to detect and combat hip
dysplasia. But only in Germany and several other European countries was it
attempted to eliminate CHD through selection, by excluding the dogs
exhibitting this condition from breeding in the populations of the different
breeds. These genetic measures, however, proved fruit-less over a period of
fore decades. The Verband für das Deutsche Hundewesen (VDH) (German Kennel
Club) failed in its attempts to gain approval for this initiative from the
American Kennel Club (AKC), The Kennel Club (KC) or the Société Centrale
Canine (SCC) in France.
The incidence of hip dysplasia still persists at around 60 - 65 % in all the
canine populations of the Western countries. When other pathological changes
are included, the total skeletal morbidity rate is as high as 70 - 75%. In
most cases HD is accompanied by other skeletal diseases.
Veterinarians in the Western countries have traditionally advocated a
»balanced diet« – with-out specifying further details – and have left it to
the animal feedstuffs industry to provide dogs with »optimized complete
diets«. At present, around 80 – 90 % of the total canine population is
being fed completely or partially with industrially manufactured
preformulated food, which has consequently become accepted as »healthy dog
food«. These new feeding methods, however, have been unable to signify-cantly
improve morbidity in terms of canine hip dysplasia. Indeed, there is also a
very high total morbiddity level of numerous nutritional diseases affecting
various organ systems.
Marc Torel and Klaus Dieter Kammerer – a veterinarian and a Pharmamanager
with medical training – believe that breeding programs and industrially
produced dog food in its present form cannot hope to bring about any
fundamental improvements in the incidence of canine hip dysplasia because
CHD is not heritable and because existing dog food does not prevent, but is
in fact the original cause of CHD. In these authors view, canine hip
dysplasia is induced solely by malnutrition.
An article in the TU caused a worldwide sensation
In 1996, the respected German journal »Tierärztliche Umschau« (TU)
(Veterinary Review) published the continuation article »Topical notes on
canine hip dysplasia« by Marc Torel and Klaus Dieter Kammerer, in which
these authors traced the entire history of the development of canine hip
dysplasia. They claimed that the hereditary nature of CHD had never been
conclusively proven and is not supported by any objective evidence, and
that breeding programs over three decades were thus inevitably doomed to
failure (Tierärztliche Umschau, Volume 51, pp. 455 ff., 1996).
In the opinion of Torel/Kammerer, everything points to the probability that
CHD has an alimentary/ hormonal etiology and pathogenesis associated with
malnutrition and increased production of somatotropin, tri-iodothyronine (T3),
thyroxine (T4), parat-hormone and insulin-like growth factor IGF-I
in the canine organism. The authors went on to draw conclusions regarding
the nutrition and keeping of dogs and gave recommendations for the
prevention of their skeletal diseases.
The essence of their argument is that CHD is of nutritional and hormonal
origin. Malnutrition causes increased production of growth hormone, the
thyroid hormones tri-iodothyronine and thyroxine, parat-hormone and
insulin-like growth factor in dogs.
The authors give a detailed analysis of the malnutrition and its adverse
skeletal effects.
The book aroused great interest among veterinarians and met with a worldwide
response. The authors and the editors of TU have received consistently
positive feedback
from the USA, the whole of Europe, South Africa and
Australia.
The Thirty Years‘ War and its bitter struggles
These sensational »Topical notes« formed the basis for the Compendium
»The Thirty Years‘ War 1966-1996«
published in 1997. The book, described with a certain selfirony by the
authors Torel/Kammerer as a controversial pamphlet, provided further data,
facts and back-ground on the subject of canine hip dysplasia, especially in
the expanded and revised 2nd edition published in March 1999 (ISBN
3-9807236-1-5).
The title is a deliberate reference to the religious war of 1618-1648 in
Central Europe, since the at-tempts to combat hip dysplasia in the
associations and societies, as described by the authors with more than a
touch of irony, were not only characterized by the ferocity of a military
campaign but, in the face of prevailing dogmatism, were also ultimately
futile.
With its »furor teutonicus« – its teutonic fury – the Compendium created an
uproar. The authors Torel/Kammerer were not only skilled in verbal
cut-and-thrust when deploying their arguments, but also brought up heavy
artillery to support their attack. The hostilities, with their attacks and
counter-attacks between the authors and their opponents in industry and
veterinary medicine, are a cause of ongoing concern to various official
agencies. The Bundestierärztekammer (BTK) (Federal German Veterinary
Authority), the VDH (German Kennel Club) and the Waltham/Effem/Masterfoods
company boycotted the book on the basis of cartel agreements and suppressed
reviews and reports in the veterinary literature, the societies and the
media.
The Error of the Millennium in Veterinary Medicine
One year later, on 10 Mar. 2000, Klaus Dieter Kammerer published his new
book entitled
»The Error of the Millennium in Veterinary Medicine«
subtitled
»Malnutrition-induced Hip Dysplasia as a
Non-hereditary Skeletal Disease of Dogs«
In his new book K.D. Kammerer traces, with a restrained style of
presentation and including much new informa-tion,
the entire development of canine diet over the past thousand years up to the
end of 1999, as well as the nu-trition-related
skeletal diseases affecting dogs.
The »trio infernale« have no alternative but to diffame and attempt to
completely discredit the authors Torel/Kammerer, whose charges, if proved
correct, would disgrace and compromise in an unprecedented manner the
leading figures of canine science, the veterinary profession and animal
feedstuffs industry for their blundering and charlatanism continuing over
decades. The Compendium »The Thirty Years‘ War« and the Compendium »The
Error of the Millennium in Veterinary Medicine« has become a full-blown
political issue.
The author piles fact upon fact to create an impressive, many coloured
mosaic that ultimately portrays a dismal scenario:
The 12 theses of Klaus
Dieter Kammerer:
1. As in all the Western countries, about 80 – 85 % of the approximately
5.5 million dogs in the Federal Republic of Germany are suffering from more
or less chronic or subchronic obesity, liver parenchymal damage, metabolic
diseases, gastrointestinal disorders with pancreatic insufficiency,
cardiovascular diseases with vascular sclerosis, impaired immune system with
an increased incidence of allergies, infections and carcinomas, as well as
a variety of disorders of the musculoskeletal system including dysplasias of
all joints, but especially the hip. The life expectancy of dogs is
considerably reduced, with cancer now heading the cause of death statistics.
2. Similarly, 80 – 85 % of dogs are nourished completely or partially with
industrially produced ready
made food which, apart from the quality deficits of the raw materials,
generally also suffers from methodological errors in its formulation and
manufacturing process. Morbidity due to the aforementioned diseases over the
last 30 years has shown an unmistaken able correlation with the sales of the
animals feedstuffs industry. Especially the skeletal diseases with hip dysplasia are directly related to the decades of mal-nutrition to which dogs
have been subjected and are their immediate consequence.
3. The principle applied in the manufacture of modern dog food was adopted
from the mass production of domestic and slaughter animals, especially calf
and pig fattening. Calves and piglets are fed a mixture of the primary
nutrients
carbohydrates, proteins, fats,
minerals and vitamins
calculated to induce rapid growth and weight gain. For retail trade
purposes, the 25 and 50 kg bags of calf and pig food were repackaged for
dogs into small packs and cans with a water con-tent of up to 80 %. This
food differs from slaughter animal feed only by the inclusion of synthetic
aromatizers, flavouring agents and attractants specific to dogs, and by the
elaborate package-ing. One further difference, however, is that to dupe
consumers dog food also contains soya designed – sometimes into the very
fibre structures - to simulate meat and »meaty lumps«. The genetic
manipulation of soya results in lipid deposits in the organs and the content
of phytoestrogens leads to pathological changes in the skeleton:
»Frankenstein Food«
So not only the alleged heritability of CHD has been adopted from domestic
and slaughter ani-mal breeding, but also its nutritional basis.
4. Heating of the raw materials with pressurized steam to as much as 250
°C
and the subsequent hotair drying breaks down the carbohydrates, but also
denatures the proteins and fats and destroys all the natural vitamins. Also
destroyed or altered are the secondary nutrients and bioactive substances
vital for longterm animal health. A standardized mineral and vitamin premix
is therefore added, but usually in excess. The phytoestrogens remain largely
intact, however, and exert the same effects on the body, including bone
metabolism, as endogenously produced sex hormones, while the lipid deposits
lead to sclerosis of the blood vessels and thus to hypertension, cardiac and
circulatory diseases. These feeding methods in dogs lead especially to
hyperalimentation, fibrous osteodystrophy, osteochondrosis (OCD), hormonal
dysfunctions as well as vitamin A and D3 hypervitaminosis
with their associated pathological skeletal changes including hip dysplasia.
Vitamin D3 overdosage alone, both in food and vitaminized mineral
preparations, can induce Legg
Calvé-Perthes disease with aseptic necrosis of the femoral head with
mush-room-shaped and cylindrical protrusions and consequently CHD. Most
ready-made food usually has added appetizers that usually stimulate
consumption, resulting in more or less marked obesity and, in the long term,
to chronic diseases of various organ systems, especially of the
cardio-vascular type. Modern domestic animal food designed in the laboratory
became causally responsible for hip dysplasia in dogs. Whereas calves and
pigs, after reaching a certain size and weight within a few months, are
slaughtered together with their pathologically altered skeleton, dogs have
to propel themselves around on their ruined bones for the rest of their
lives.
5. The heritability of hip dysplasia and the other skeletal diseases of
dogs has never been demonstrated. The first veterinarians to investigate the
etiology of CHD elaborated a number of hypotheses of which multifactorial
inheritance was finally chosen as the winning candidate, since it appeared
to explain everything. Subsequent investigators adopted these assumptions
unreflectingly and uncritically and merely perpetuated the error. Others
wrote papers designed to please their sponsors, for remuneration. There is a
certain ironic pleasure to be had from the fact that Professor Helmut Meyer,
as a nutritional scientist, Senior Assistant and prospective Director of the
Institute of Animal Nutrition of Hanover Veterinary College published a
paper on the heritability of CHD in 1968, although this was not his
specialization and he had no under-standing of canine genetics, but was
already maintaining very close and financially rewarding business links with
Waltham/Effem/Masterfoods. Finally, a downright dogma was established
regarding the multifactorial (polygenic) heritability of hip dysplasia.
6. It is perhaps one of the ironies of fate that CHD is indeed a
multifactorial disease, with a variety of nutrition-induced underlying
disorders as possible causative factors:
·
Hormonal dysfunctions (IGF-I, T3, T4, Parathormon,
Estrogene)
·
Vitamin A+D3+K3 - Hypervitaminosis (toxic
overdosage)
·
Rickets
(softening of bone due to Ca and vitamin D3 deficience)
·
Moeller-Barlow disease (scurvy due to vitamin C deficiency)
·
Legg-Calvé-Perthes
disease (femoral head necrosis)
·
Genu valgum
(knock-knees)
·
Fibrous
osteodystrophy (bone dystrophy)
·
Osteochondrotic syndrom (degenerative cartilage disease, OCD)
·
Hypertrophe
Osteodystrophy (HOD)
·
Obesity
(overweight,
Overload during movement
The metabolic derangements with alimentary/hormonal dysfunctions underlying
these pathologic conditions thus lead to dysplasias of the entire skeleton
and range from very mild and often clinically undiagnosed disease courses to
severe deformations. Since the dynamic and static forces involved in
movement are most pronounced at the hip joint, this is where the first
deformations (dysplasias) inevitably develop. Dysplasi-as can also occur in
all other joints, however, and they would be observed in more or less severe
form in all dogs with CHD if X-ray examination were not confined to the hip
joints. Many cases of canine hip dysplasia are also accompanied by dysplasia
of the shoulder, elbow and knee joints.
7. The etiology and pathogenesis of hip dysplasia can thus only be
explained in terms of multifactoriality, but omitting the genetic factor.
Accordingly, hip dysplasia is to be regarded as a complication of metabolic
disease in puppies and young dogs, and as a symptom of a generalized,
alimentary/hormonal skeletal disease, and on no account as a hereditary,
isolated anomaly of acetabulum and femur.
8. The multinational corporations Nestlé (Alpo, Fancy Feast, Friskies,
Migthy Dog), Colgate Palm-olive (Hill’s Science Diet), Procter & Gamble (Iams,
Eukanuba), Heinz (Amore, Gravy train, Recipe, Verts) and especially Mars
with its affiliates Waltham and Effem (Advance, Cesar, Chappy, Formula,
Frolic, Kal Kan, Mealtime, Pedigree ) control the multi-billion-dollar world
market for industrially produced dog and cat food, including 90 % of the
European market. Ralston Purina (Pro Plan, Purina) and Royal Canin
(Selection, Size mini-medium-maxi) also enjoy a certain prominence. Effem is
the market leader in Germany with a 45 – 50 % share of a market worth 3,500
million German Marks and, with a turnover of about 2,000 million German
Marks makes a profit of almost 1,000 million. These big corporations are
behind most of the many small producers.
9. These business empires have virtually unlimited funds for advertising.
In the Western countries, they have advertising budgets totaling more than
10,000 million DM to splash out on dog food advertising. In the Federal
Republic of Germany alone, Mars together with Waltham and Effem invest
almost 500 million DM annually in the public and non-public domains. The
money is de- voted firstly to poster and press adverti-sing, TV spots and
for paid public relations articles in newspapers and magazines. The canine
popular press is completely under the control of the animal feedstuffs industry
and is only allowed to publish articles favourable to its backers‘
interests. Canine science societies and associations and their functionaries
enjoy considerable financial benefits. The VDH (German Kennel Club) and its
board members are annual beneficiaries to the tune of several millions and
as such are to be regarded as completely dependent and corrupt. While all
this money is paid to buy people and votes, hush money is paid to the
parties, institutions and the judiciary.
10. The veterinary profession too is now firmly in thrall to these
multinational corporations. Many
veterinarians owe their living to this mutual sleaze and corruption. Of the
20,000 practising veterinarians in the Federal Republic of Germany, about
20-25% are unemployed anyway and another 20-25 % would have to shut up shop
if they didn’t receive the average Euro 20,000 – 25,000 annually from the
sale of special diet feeds and the X-ray system of the VDH (German Kennel
Club) they need to cover their practice costs (rent, staff). Considerable
benefits in cash and kind find their way to students of veterinary medicine,
medical-technical assistant personnel and the veterinarians themselves. The
opinion-leading veterinarians and nutritional scientists at the institutes
of animal nutrition are almost without exception bribed and travel around
the world at the expense of the various companies. They therefore recommend
these products from sheer self-interest and regale dog owners with the
benefits of industri-al dog food while keeping quiet about the
disadvantages. Since the entire advertising budgets are funded from the
turnover and hence from the sales prices of the various brands, consumers
and dog owners are actually pay-ing out of their own pockets for the
brainwashing to which they are continually subjected, not even realizing
that these by no means so optimized products are also grossly overpriced.
The same or similar conditions prevail in the other countries.
11. The companies mentioned above – but especially Waltham/Effem/Masterfoods
– initiated the
dogma of the heritability of skeletal diseases of dogs to conceal the
methodological errors being perpetrated in industrial animal feedstuffs
production and have sustained their campaign over three decades. Almost all
the authoritative veterinarians in the USA, UK and Germany who are or have
been involved in studying CHD have at some time received inducements from
Walt-ham/Effem/Masterfoods, Ralston Purina or the others. The financial
calculations and marketing strategies require not healthy, but sick dogs.
The diseases of the various organ systems induced by malnutrition gave the
impetus for market innovation in the form of the numerous dietary products
sold through veterinarians, to whom the industry not only delivers the
various brands free of charge, but also the sick dogs necessary for their
consumption. In this thoroughly corrupt system, the industry and veterinary
profession in the Western countries are actively engaged in white collar and
white coat crime and are perpetrating the biggest fraud in the history of
veterinary medicine, nothing less than a chronique scandaleuse.
12. The malnutrition of dogs and the dogma of the heritability of its hip
dysplasia are firstly a double
error and
considering the disaster for dogs‘ health created and sustained
over four decades by the industry and the professors in their service at the
faculties of veterinary medicine, especially in the USA, UK and Germany, can
only be described as the error of the millennium in veterinary medicine:
millions of dogs around the world have been fed to a state of illness or to
death during this period. By reforming canine nutrition, improving the
quality and eliminating the methodological errors in production, morbidity
due to the numerous nutrition-induced diseases of dogs could be
significantly lowered. In particular, the skeletal diseases associated with
hip dys-plasia could be at least considerably reduced.
This book review is protected by copyright. All rights reserved. No part of this
review may be reproduced, stored in information retrieval systems,
or transmitted, in any for or by means of electronic, mechanical,
photocopying, microfilm – without the prior written permission of the
publisher. The copy-right is held by Transanimal and Klaus Dieter Kammerer.
Version of 01.11.2006
Klaus Dieter Kammerer
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